Sudden Infant Death Syndrome Has Finally Been Solved and Cancers Are Explained As Due to the Transfer of DNA from a Drug Resistant Bacteria, Helicobacter Plyori, which was classified in 1995 as a Carcinogen, by the NIH!

(PRWEB) May 29, 2004

(PRWEB) May 29, 2004 The riddle of SIDS and what causes cancers has been solved. Â?Both cancers and Â?Sudden Infant Death SyndromeÂ?(SIDS) are caused by the same type of rapidly acting DNA, from the Helicobacter plyori bacteria,Â? according to Bruce D McKay, an independent researcher in Tampa, Florida.

Mr McKay says Â?there was a bacteria put on a list of carcinogenic agents in 1995, because it was proven to be somehow able to start two gastric stomach cancers. This was fully certified by a slect panel from the National Institutes of Health, and published on page 1621 of Science, on March 17, 1995.(1) No one before has ever explained it. The Helicobacter pylori are a Â?drug resistant speciesÂ? and frequently found to have many links to various cases of SIDS. According to McKay, no one explained it because almost everyone wrongly ‘thinks of drug resistance’ as being due to evolution, and the whole system of logic all ‘faces’ along side that concept. The real facts of the matter ‘face in the opposite direction;’ the drug resistant bacteria have the ability to trasfer their own drug resistance genes. Theose genes are passed to other cells in what is known as plasmid DNA’s.

What is also NOT well known is that Â?virtually every antibacterial drug in use can be rendered ineffective by one or another R plasmid, and many of them contain multiple drug-resistant determinants.Â?Â?(2) McKay says, Â?this is then the real Â?missing answerÂ? as to what causes both two different gastric cancers, and why many children frequently die of SIDS.” It also explains every form of Â?drug resistant cancer,Â? and all the tumors.

“Look at it in just the opposite manner,” McKay says. “There are those who do a great deal of research on transferrable DNAÂ?s every day – while the exact nature of a secondary form of DNA has never caught anyone’s attention. The DNA responsible, McKay says, should be called Â?TransDNA.Â? Researchers have studied every aspect of it – without ever coming to grips with the reality of it; it is a form of DNA that explains how the Â?properties of bacterial cells change at a frequecy that is much higher than that expected on the basis of spontaneous mutations, and the properties affected often include several of medical importance, relating to antigenicity, virulence, and adhesion.’(3) In other words Â? it explans that something has got to be wrong with how researchers have all been looking at each of these various processes.”

McKay says a TransDNA system fits the ‘flip-flop’ of some systems in DNA. It fits as ‘altering the range of host bacterial cells,’ when they become infected by phanges. What researchers failed to account for was Â?the similarity between two clases of site-specific recombination events,Â? it fits that too; and those events were reported as Â?not superficial.Â?(4) In brief it fits many different changes taking place within a drug resistant bacteria’s transferable DNA. He says, this form of DNA can even account for the rapid switching of ‘invertible DNA segments.’ It is an efficient and almost instant mechanism for adaptaion to any change in environment. It ‘sets up’ and can even ‘reset,’ the actual combinations of ‘virulence and antibiotic-resistance genes’ that it merges together to form a single plasmid. And it even fixes Â?the rearrangement of genes into very complex plasmidsÂ?Â?Â?(5) McKay says, all of these things happen to the DNA of drug resistant bacteria, and no one has ever before recognized how all of this is a part of a common DNA system of transfer, regulation, and control. Furthermore, he feels that anyone who attribute all of these effects to evolution, is constantly ‘turning away’ from the very thing that everyone has been looking for – and that’s exactly what’s been going on.

If a drug or some other highly disruptive factor hits, altering the normal environment of the drug resistant bacteria, they counter by immediately switching genes. McKay points out, they would all be dead, head on with just about any problem, if they relied on an evolution based reaction. The antibiotics would all work – if they drug resistant bacteria and the drug resistant cancer and tumor cells, were all running on evolution! And the DNA that is passed from the drug resistant bacteraia can even generate a change of its common adhesion molecules, as another very easy way to survive. Meanwhile, researchers who very seriously study metastasis, laborously following so-called cancer causing cells as they metastise to various tissues in the body – while trying to deduce how in the world the cancer cells can adhere or just plain stick to this or that given type of cell, where they lodge and then cause the next cancer to take place.    

If the transferable plasmid with the replication modules from a drug resistant bacteria, (i.e., such as the very small, Helicobacteria pylori,) is able to Â?sets the stageÂ? within a new host, isnÂ?t that what readily allow the highly virulent bacteria to come in, attack, and then readily replicate in terms of their new environment? So how does SIDS get started? It is transmitted into a young child from a parent, relative, pastor, baby sitter, or a friend – in a direct parallel to the early 1900Â?s when certain persons were first recognized as TB carriers!Â?

According to McKay, many cancers start in the very same way! But there are very few persons who can even imagine the transfer of drug resistant genes as easily crossing species lines Â? but it’s been proven – that is exactly what they do! What causes DNA segements to move, flip flop, change, or become Â?mobilized?Â? Such things never get a fair answer. McKay says, Â?itÂ?s the TransDNA at work when the plasmid DNA is being generated. Researchers have studied the most dangerous ones, which are those coming from bacteria that replicate a double stranded DNA. ThatÂ?s the ones that can readily pass into human cells. McKay says this even explains why Helictobacter pyori, a bacteria, has been put on a list of carcinogens!

Why do cancer cells become Â?neoplastic?Â? Evolution is faced down once again! Some of the DNAÂ?s in the drug-resistance (R) plasmids are able to change each time they replicate. This is known as “relaxed replication.” It also causes the bacteriaÂ?s drug-resistance genes, to become quite plastic, just as the cells and the same transferable DNA from bacteria can also account for the Â?neoplasticÂ? nature of almost every cancer cell line. Serveral resistance genes may be carried (and/or transferred) by a single R plasmid, and the colicinogenic (Col) plasmids can also encode for some very small proteins (called colicins,) that kill a variety of other bacteria that may become dangerous to the producing organism. Hence, when cancer cells end up with the colicinogenic plasmid DNA modules being in them, that DNA can even encode Â?an immunity protein,Â? which gives ever indication that these cells they will never commit suicide, and so they appear as if they will never, ever die!

Will you read all about this in any of todayÂ?s scientific journal? If everyone has been taught to think that it all works just the other way around, it will take a truly top notch scientific journal to publish this. Possibly, but not in the near future. And then again, since SIDS strikes as it does Â? and it has been shown to be due to the virulence built into the DNAÂ?s that are produced and transferred Â? it won’t be long before someone out there is going to take full notice of all this. The proteins being ‘set up are encoded’ are very important for the colonization of the bacteria in the new host, regardless of how the TransDNA introduces these factors. In terms of Helicobacter pylori, the toxins are what allows the admittance of these bacteria into the stomach, and then the counter-evolution, cryptically introduced virulence plasmids may prove to be overwhelming. The unfortunate small children, too young to be told anything about evolution (or… cancers) is so quickly overwhelmed with a toxic reaction moving so fast that there isn’t even any tell-tale scars left behind!

Maybe based on that noted above, and knowing about how TB is passed by a carrier, some person or persons in the medical community will finally catch on. McKay says, even smoke partiles can carry cancer causing DNA’s from tobacco associated bacteria, in much the same way. Hopefully, you, as the reader, will begin to realize that the TransDNAÂ?s are there and that they are initializing every cancer and every tumor. They normally direct an entire unseen system of DNA control throughout the bacterial kingdom. What am I trying to pull? Why doesnÂ?t someone out there just stop and recognize that TransDNA systems do exist! Is it true that DNA is all the same? Or is there a chance that there is a very light, colorless, and almost perfectly clear, distinct type of DNA, frequently involved in whatÂ?s behind the flip-flop of systems, or acting as an unseen switch between two alternate DNA forms, or as inverting key DNA, or otherwise jumping jeans, or being involved in the process mobilizations, or prospering DNA transpons to emerge?

Well if there is a DNA like this that exists, and it is able to rapidly move and yet maintain a spontaneously transferring form, would it not be the counterpart to the intrinsically slower, easier to see, ‘fatterÂ? and much more dense forms of DNA? If so, wouldnÂ?t it turn out that WHAT we do NOW know about DNA is simply the Â?building blocksÂ? of mother nature. And the other is actually a primary form of DNA, and it has carried on all the minute changes and otherwise directed and even redirected the operational nature of everything, all along! Understandably, the way that this problem has always been faced, has left every truly knowledgeable person with little more than a series of articles published, and for more than half a century we have tried, with many millions and even billions having been spent on the task. Add to that, anyone is thought to know nothing at all if they are of the opinion that the whole problem faces in the other direction – and bacteria are able to actually transfer a form of DNA – and that’s whatÂ?s truly involved in starting each new cancer or tumor, or so says this modern day Christopher Columbus! But if all of this on drug resistance is behind an illusion in people’s minds Â? then this readily explains why everyone has been working for decades, doing a million and one studies and countless Â?approved investigationsÂ? on all types of cells, cancers, tumors, DNAÂ?s, plasmids, and every aspect of drug resistant diseases!

Mr. McKay respectfully suggests therefore, that itÂ?s time for everyone to take a fresh look. As one researcher put it, the “assumptions behind today’s medicine are left over from the turn of the century, when science was forcing dogmatic religion to see the evidences of evolution.”(6) If it is basically an illusion, then what almost every member of the medical community thinks of all the antibiotics as somehow Â?loosing their effectivenessÂ? is in keeping with that illusion posed view! If you get to this point in addressing the logic, next ask yourself why none of them can ever tell you – why the whole bunch of antibiotics that were all once so higly effective, now have no punch at all!”

McKay says this is the real problem that “the medical community needs to come to grips with. It’s either true that bacteria are able to overcome antibiotics by their own TransDNA activities, and this is why they have been doing all along, or it’s not true at all. (Most can look you in the eye and respond, when theyÂ?re thinking evolution as if its wrong to think or believe that bacteria do transfer DNA.) So just tell them THE PROOF OF THE MATTER is that researchers have found when Â?patients are given oral tetracycline, the predominant fecal E. coli isolates now show up with tetracycline-resistance R plasmids – within 1 week! Tell them this also. The antibiotics used widely as growth promothers in lifestock have also been shown to transfer readily from animal to humans.Â?(7) Then ask… is all this a good clean, general case of survival of the fittest or not?

According to McKay, itÂ?s not – simply because there is an extensive time frame needed for such things as this to happen under the guise and dictates of evolution! In other words, those in the medical community and the sciences have got it all wrong! There is a TransDNAÂ?s and it typically exchange gnes, and DNA’s, and this DNA can even transfer the DNA modules needed to transfer it self at some future date, while existing as an all but invisible Â? very light and wispy form of almost perfectly clear DNA that canÂ?t even be picked up or sensed on an electron microscope Â? because the electrons fly right through it! Yes, this form of DNA is so clear, that it is virtually, Â?microscopically undetectable!Â? What can be seen of it is only a shadow, when metal staining techniques are used – yet it accounts for cancers and the deadly toxins that bring about SIDS.

Â?The one ‘other way’ it works,” he says, “is that some strains of these same drug resistant bacterium are also able to replicate their various drug resistant genes – and then they are able to transfer that “drug resistance” (i.e. the drug resistance genes) into a human cell. After a while, those DNA’s flip flop, or they otherwise change places, and then then those same drug resistant genes end up (i.e., being found later,) as the driving force behind amost every form of cancer and every tumor! Brinton has even proven that the transferrable DNAÂ?s need NO chromosomes present to do spontanelusly tranfer. And when they do transfer they do not even need the bacteria being present from which they originate!(8) According to McKay, Â?when the self-transferring DNA modules needed for self-transferring are not included, that accounts equally as well for the charastices that we commonly refer to as (non-malignant) tumors.”

McKay – ordinary cells in a given tissue have the same TransDNA system working in them too! The research community never gained sight of this,Â? he says. Â?They were thinking that all of the above was evolution(!), and in 1981, Griffin and his associates noted that there was a great Â?heterogeneityÂ? among normal cells after they were dispersed from one another and grown in culture.Â?(9) McKay says, Â?What they found is that the cells they were studying would suddenly take on a totally independent nature or an entirely differing manner of activity(!) each time they were broken apart and placed in a culture! This is simply because thatÂ?s when they would loose the Â?TransDNA mechanism,Â? that normally links everything up that is moving from cell to cell.

After the resulting flip-flops, and other changes that took placeÂ? as he explains it, Â?then the cells do not act normally simply because the TransDNA system linking one cell to another cell is no longer in operation. All that was readily lost when the very delicate microtubules that bridge from one cell’s membrane proteins to another were disrupted or broken, and the cells were otherwise torn apart. That is what consistently happen every time normal cells were put into a culture to comapre them to cancer cells. Almost every study of cells has been seriously hampered, simply as a result of what happens when anyone places cells in almost any culture medium.”

Â?Have we all been unknowingly brainwashed or what? Go and look in any good biology text book and find about a model of the cell. Now look closely at the cellÂ?s ‘membrane proteins.’ Just below that you should find some tiny little microtubules running down to the very heart of the cell, to the chromosomes and the cellÂ?s DNA. Now go back up where the microtubules meet at the membrane protein. That, my friend, is where the microtubules should pass out of the cell. And from there they should bridge to the next cell’s membrane protein. It it was all explained right there, that this is what Â?hooks togetherÂ? all the cells in a given tissue, at the DNA level Â? you would be able to read all about it, right there Â? but you canÂ?t. And so this readily proves the whole thing! In other words, to correct ‘the static nature of the cell theory,’ the third statement of the Cell Theory sould actually be something like, ‘The cells that make up any given tissue, are connected by almost invisible strings of very fine microtubules, to all the other cells, and this provides each given tissue with a highly specific network of intercooperative control at the DNA level.’”

Â?In additon, all this about cells starting to act independently when placed in a culture, rapidly explains what has always hindered and hampered all of the medical research which has ever been done, on almost any gene-related diseases, or any form of cancer.Â? McKay says, Â?It is highly unfortunate, but the researchers were just not comparing apples to apples!Â? He says, Â?it also explains what causes the drug resistance genes to emerge as they do, in almost every cancer and every tumor – regardless of the tissue or origin. And it as well explains, he says, what lays just behind many other diseases such as MS, MD, Diabetes, AIDS, HIV, and a wide assortment of other gene-related diseases,Â? according to McKay. In that regard McKay belives that Â?all of those diseases would be just like cancers, if the DNA that initialized them had been just a little more complex. In other words, itÂ?s the double stranded DNA modules that cause the cancers and tumors, but if the the self-transfer DNA modules are not included when all these other disease were first initiated, they are only ‘various types,’ of lesser activity and various types of cell disturbances – because the well hidden TransDNA mechanism is an intrinsic part of all nature.Â?

There is also evidence and photographs of microtubules linking cells to cells, and is available for further study, analysis, and evaluation, on the net at McKayÂ?s web page – thecancerdiscovery.net . The photographic evidence included in actual photos, show an otherwise all but invisible interlinking of microtubules that run between cells, by Professor Mitsuo Yamanaka in Shikoku Japan, the Department of Biology, Faculty of Science at Kochi University, Japan. Professor Yamanaka’s evidently scanned cells that were yet together as a tissue, as indicated by this photograph… http://www.is.kochi-u.ac.jp/Bio/cellbio/welcome.html

“Nor are the findings that surround the Â?TransDNAÂ? as a spontaneous cancer causing recombinant DNA, difficult to comprehend, or explain. Everything begins to fall into place,” according to McKay. Â?The major problem has always been that the TransDNA being passed or transferred into the cell is so light that the electrons from the electron microscope pass right through it. The scientists have even classified it as an r-plasmid, or a form of ‘resistance gene’ transferring DNA Â? without ever making the realization that it was not just some kind of orphaned thing. But thatÂ?s partly because just a shadow of could ever be seen, and even then it took high speed photography and “metal staining” just to see the shadow of it Â? and they think everything they see floating around out there is all due to the process of evolution. No one ever realized it was part of the a super large, spontaneous control system, so extensive and so immense that it actually regulates the ongoing DNA activity of almost all of the entire body of nature! What was going on, in other words, was far to big for anyone to see!

This briefly explains how “drug resistance genes” end up as suddenly emerging in cancers and tumor cells, and why the spontaneously moving form of recombinant DNA from bacteria causes sudden Infant Death Syndrome(SIDS)and Cancers.

___________

1. Thompkins, Lucy S., and Falkow, Stanley, Â?The New Path to Preventing Ulcers,Â? Science, Vol 267, 17 March 1955, page 1621.

2. Joklik, K. Wolfgang, Willett, Hilda P. Amos, D. Bernard, and Wilfert, Catherine M., Zinsser Microbiology, 19th Edition, Joklik, Wolfgang K., ed., Appleton & Lange, California, 1988, pg 122,

3.-5. Ibid., #2., page 121.

6. Becker, Robert O., and Selden, Gary, The Body Electric, Quill, New York, 1985, page 20.

7. Ibid., #2., page 124.

8. Becker, Robert O., and Selden, Gary, The Body Electric, Quill, New York, 1985, page 20.

9. Griffin, J. E., Altman, D. R., Durant, J. L. and Wilson, J. D., “Variation in steroid 5a-reductase activity in cloned human skin fibroblasts,” J. Biol. Chem., 256: 3662-3666, 1981.

For More Information Please Contact

Bruce D. McKay, Epistemologist

11308 N. Hamner Ave

Tampa, FL USA 33612

(813) 933-4905

Web Page: thecancerdiscovery.net

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